{"product_id":"cardiovascular-physiology-mosby-physiology-monograph-series-paperback-9780323594844","title":"Cardiovascular Physiology; Mosby Physiology Monograph Series (Paperback \/ softback) 9780323594844","description":"\u003cfont face=\"Georgia\"\u003e\r\n\u003cp\u003e\u003cfont size=\"6\"\u003eCardiovascular Physiology\u003c\/font\u003e\u003cbr\u003e\r\n\u003cfont size=\"5\"\u003eMosby Physiology Monograph Series\u003c\/font\u003e\u003c\/p\u003e\r\n\r\n\u003cp\u003e\u003cem\u003e\u003cp\u003eMaster physiologic principles of the cardiovascular system\u003c\/p\u003e\u003c\/em\u003e\u003c\/p\u003e\r\n\r\n\r\n\u003cp\u003e\u003cfont size=\"4\"\u003eAchilles J. Pappano (Author), Withrow Gil Wier (Author)\u003c\/font\u003e\u003c\/p\u003e\r\n\r\n\u003cp\u003e\u003cfont size=\"3\"\u003e9780323594844, Elsevier Health Sciences\u003c\/font\u003e\u003c\/p\u003e\r\n\r\n\u003cp\u003e\u003cfont size=\"3\"\u003ePaperback \/ softback, published 10 January 2019\u003c\/font\u003e\u003c\/p\u003e\r\n\r\n\u003cp\u003e\u003cfont size=\"3\"\u003e300 pages, 230 illustrations (230 in full color)\u003cbr\u003e23.4 x 19 x 2 cm, 0.63 kg\u003c\/font\u003e\u003c\/p\u003e\r\n\r\n\r\n\r\n\r\n\r\n\u003cp align=\"justify\"\u003e\u003cstrong\u003e\u003cfont size=\"3\"\u003e\u003cp\u003eGain \u003cb\u003ea foundational understanding of cardiovascular physiology \u003c\/b\u003eand how the cardiovascular system functions in health and disease. \u003cb\u003eCardiovascular Physiology\u003c\/b\u003e, a volume in the \u003ci\u003eMosby Physiology Series, \u003c\/i\u003eexplains the fundamentals of this complex subject in a clear and concise manner, while helping you \u003cb\u003ebridge the gap between normal function and disease\u003c\/b\u003e with pathophysiology content throughout the book.\u003c\/p\u003e\n\u003cul\u003e \u003cli\u003eHelps you\u003cb\u003e easily master the material in a systems-based curriculum \u003c\/b\u003ewith learning objectives, Clinical Concept boxes, highlighted key words and concepts, chapter summaries, self-study questions, and a comprehensive exam to help prepare for USMLEs.\u003c\/li\u003e \u003cli\u003eKeeps you current with\u003cb\u003e the latest concepts in vascular, molecular, and cellular biology \u003c\/b\u003eas they apply to cardiovascular function, thanks to molecular commentaries in each chapter.\u003c\/li\u003e \u003cli\u003eIncludes \u003cb\u003eclear, 2-color diagrams \u003c\/b\u003ethat simplify complex concepts.\u003c\/li\u003e \u003cli\u003eFeatures\u003cb\u003e \u003c\/b\u003eclinical commentaries that show you\u003cb\u003e how to apply what you've learned \u003c\/b\u003eto real-life clinical situations.\u003c\/li\u003e \u003cli\u003e\n\u003cb\u003eEnhanced eBook version included with purchase.\u003c\/b\u003e Your enhanced eBook allows you to access all of the text, figures, and references from the book on a variety of devices.\u003c\/li\u003e \u003c\/ul\u003e \u003cb\u003e  \u003c\/b\u003e\u003cp\u003eComplete the Mosby Physiology Series! Systems-based and portable, these titles are ideal for integrated programs.\u003c\/p\u003e  \u003cul\u003e  \u003cul\u003e  \u003cp\u003e \u003c\/p\u003e\n\u003cli\u003eBlaustein, Kao, \u0026amp; Matteson: Cellular Physiology and Neurophysiology\u003c\/li\u003e \u003cli\u003eCloutier: Respiratory Physiology\u003c\/li\u003e \u003cli\u003eKoeppen \u0026amp; Stanton: Renal Physiology\u003c\/li\u003e \u003cli\u003eJohnson: Gastrointestinal Physiology\u003c\/li\u003e \u003cli\u003eWhite, Harrison, \u0026amp; Mehlmann: Endocrine and Reproductive Physiology\u003c\/li\u003e \u003cli\u003eHudnall: Hematology: A Pathophysiologic Approach\u003c\/li\u003e\n\u003c\/ul\u003e\n\u003c\/ul\u003e\n\u003col\u003e \u003cli\u003e Appendix - Comprehensive MCQ review examination\u003c\/li\u003e \u003cli\u003eKeywords\/concepts\u003c\/li\u003e \u003c\/ol\u003e\u003c\/font\u003e\u003c\/strong\u003e\u003c\/p\u003e\r\n\r\n\u003cp\u003e\u003cfont size=\"3\"\u003e\u003cp\u003e\u003cb\u003eChapter 1\u003c\/b\u003e \u003cb\u003eOVERVIEW OF THE CIRCULATION AND BLOOD\u003c\/b\u003e\u003c\/p\u003e \u003cp\u003eThe Circulatory System\u003c\/p\u003e \u003cp\u003eBlood\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eErythrocytes\u003c\/p\u003e \u003cp\u003eLeukocytes\u003c\/p\u003e \u003cp\u003eLymphocytes\u003c\/p\u003e \u003cp\u003ePlatelets\u003c\/p\u003e \u003cp\u003eBlood Is Divided into Groups by Antigens Located on Erythrocytes\u003c\/p\u003e \u003cp\u003eSummary\u003c\/p\u003e \u003cp\u003eCase 1-1\u003c\/p\u003e \u003cb\u003e \u003c\/b\u003e\u003cp\u003eChapter 2 EXCITATION: THE CARDIAC ACTION POTENTIAL\u003c\/p\u003e \u003cp\u003eCardiac Action Potentials Consist of Several Phases\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eThe Principal Types of Cardiac Action Potentials Are the Slow and Fast Types\u003c\/p\u003e \u003cp\u003eIonic Basis of the Resting Potential\u003c\/p\u003e \u003cp\u003eThe Fast Response Depends Mainly on Voltage-Dependent Sodium Channels\u003c\/p\u003e \u003cp\u003eIonic Basis of the Slow Response\u003c\/p\u003e \u003cp\u003eConduction in Cardiac Fibers Depends on Local Circuit Currents\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eConduction of the Fast Response\u003c\/p\u003e \u003cp\u003eConduction of the Slow Response\u003c\/p\u003e \u003cp\u003eCardiac Excitability Depends on the Activation and Inactivation of Specific Currents\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eFast Response\u003c\/p\u003e \u003cp\u003eSlow Response\u003c\/p\u003e \u003cp\u003eEffects of Cycle Length\u003c\/p\u003e \u003cp\u003eSummary\u003c\/p\u003e \u003cp\u003eCase 2-1\u003c\/p\u003e \u003cb\u003e \u003c\/b\u003e\u003cp\u003eChapter 3 \u003cb\u003eAUTOMATICITY: NATURAL EXCITATION OF THE HEART\u003c\/b\u003e\u003c\/p\u003e \u003cp\u003eThe Heart Generates Its Own Pacemaking Activity\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eSinoatrial Node\u003c\/p\u003e \u003cp\u003eIonic Basis of Automaticity\u003c\/p\u003e \u003cp\u003eOverdrive Suppression\u003c\/p\u003e \u003cp\u003eAtrial Conduction\u003c\/p\u003e \u003cp\u003eAtrioventricular Conduction\u003c\/p\u003e \u003cp\u003eVentricular Conduction\u003c\/p\u003e \u003cp\u003eAn Impulse Can Travel Around a Reentry Loop\u003c\/p\u003e \u003cp\u003eAfterdepolarizations Lead to Triggered Activity\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eEarly Afterdepolarizations\u003c\/p\u003e \u003cp\u003eDelayed Afterdepolarizations\u003c\/p\u003e \u003cp\u003eElectrocardiography Displays the Spread of Cardiac Excitation\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eScalar Electrocardiography\u003c\/p\u003e \u003cp\u003eDysrhythmias Occur Frequently and Constitute Important Clinical Problems\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eAltered Sinoatrial Rhythms\u003c\/p\u003e \u003cp\u003eAtrioventricular Transmission Blocks\u003c\/p\u003e \u003cp\u003ePremature Depolarizations\u003c\/p\u003e \u003cp\u003eEctopic Tachycardias\u003c\/p\u003e \u003cp\u003eFibrillation\u003c\/p\u003e \u003cp\u003eSummary\u003c\/p\u003e \u003cp\u003eCase 3-1\u003c\/p\u003e \u003cb\u003e \u003c\/b\u003e\u003cp\u003eChapter 4 \u003cb\u003eTHE CARDIAC PUMP\u003c\/b\u003e\u003c\/p\u003e \u003cp\u003eThe Microscopic and Gross Structures of the Heart \u003c\/p\u003e \u003cp\u003e\u003ci\u003eCardiac Muscle (myocardial) Cell Morphology\u003c\/i\u003e\u003c\/p\u003e \u003cp\u003eStructure of the Heart: Atria, Ventricles, and Valves\u003c\/p\u003e \u003cp\u003eThe Force of Cardiac Contraction Is Determined by Excitation-Contraction Coupling and the Initial Sarcomere Length of the Myocardial Cells\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eExcitation-Contraction Coupling Is Mediated by Calcium\u003c\/p\u003e \u003cp\u003eMechanics of Cardiac Muscle\u003c\/p\u003e \u003cp\u003eThe Sequential Contraction and Relaxation of the Atria and Ventricles Constitute the Cardiac Cycle\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eVentricular Systole\u003c\/p\u003e \u003cp\u003eEchocardiography Reveals Movement of the Ventricular Walls and of the Valves\u003c\/p\u003e \u003cp\u003eThe Two Major Heart Sounds Are Produced Mainly by Closure of the Cardiac Valves\u003c\/p\u003e \u003cp\u003eThe Pressure-Volume Relationships in the Intact Heart\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003ePassive or Diastolic Pressure-Volume Relationship\u003c\/p\u003e \u003cp\u003eActive or End-Systolic Pressure-Volume Relationship\u003c\/p\u003e \u003cp\u003ePressure and Volume during the Cardiac Cycle: The P-V Loop\u003c\/p\u003e \u003cp\u003ePreload and Afterload during the Cardiac Cycle\u003c\/p\u003e \u003cp\u003eContractility\u003c\/p\u003e \u003cp\u003eThe Fick Principle Is Used to Determine Cardiac Output\u003c\/p\u003e \u003cp\u003eMetabolism of ATP and its Relation to Mechanical Function\u003c\/p\u003e \u003cp\u003e\u003ci\u003eFatty Acid Metabolism\u003c\/i\u003e\u003c\/p\u003e \u003cp\u003eCarbohydrate Metabolism\u003c\/p\u003e \u003cp\u003eInterrelation between Fatty Acid and Carbohydrate Metabolism\u003c\/p\u003e \u003cp\u003eEffects of plasma substrate and insulin levels\u003c\/p\u003e \u003cp\u003eCardiac O\u003csub\u003e2\u003c\/sub\u003e Consumption and the Link between Ventricular Function and Cardiac Metabolism\u003c\/p\u003e \u003cp\u003eSummary\u003c\/p\u003e \u003cp\u003eCase 4-1\u003c\/p\u003e \u003cb\u003e \u003c\/b\u003e\u003cp\u003eChapter 5 \u003cb\u003eREGULATION OF THE HEARTBEAT\u003c\/b\u003e\u003c\/p\u003e \u003cp\u003eHeart Rate is Controlled Mainly by the Autonomic Nerves\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eParasympathetic Pathways\u003c\/p\u003e \u003cp\u003eSympathetic Pathways\u003c\/p\u003e \u003cp\u003eHigher Centers Also Influence Cardiac Performance\u003c\/p\u003e \u003cp\u003eHeart Rate Can Be Regulated via the Baroreceptor Reflex\u003c\/p\u003e \u003cp\u003eThe Bainbridge Reflex and Atrial Receptors Regulate Heart Rate\u003c\/p\u003e \u003cp\u003eRespiration Induces a Common Cardiac Dysrhythmia\u003c\/p\u003e \u003cp\u003eActivation of the Chemoreceptor Reflex Affects Heart Rate\u003c\/p\u003e \u003cp\u003eVentricular Receptor Reflexes Play a Minor Role in the Regulation of Heart Rate\u003c\/p\u003e \u003cp\u003eMyocardial Performance Is Regulated by Intrinsic Mechanisms\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eThe Frank-Starling Mechanism Is an Important Regulator of Myocardial Contraction Force\u003c\/p\u003e \u003cp\u003eChanges in Heart Rate Affect Contractile Force\u003c\/p\u003e \u003cp\u003eMyocardial Performance Is Regulated by Nervous and Humoral Factors\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eNervous Control\u003c\/p\u003e \u003cp\u003eCardiac Performance Is Also Regulated by Hormonal Substances\u003c\/p\u003e \u003cp\u003eSummary\u003c\/p\u003e \u003cp\u003eCase 5-1\u003c\/p\u003e \u003cb\u003e \u003c\/b\u003e\u003cp\u003eChapter 6 \u003cb\u003eHEMODYNAMICS\u003c\/b\u003e\u003c\/p\u003e \u003cp\u003eVelocity of the Bloodstream Depends on Blood Flow and Vascular Area\u003c\/p\u003e \u003cp\u003eBlood Flow Depends on the Pressure Gradient\u003c\/p\u003e \u003cp\u003eRelationship Between Pressure and Flow Depends on the Characteristics of the Conduits\u003c\/p\u003e \u003cp\u003eResistance to Flow\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eResistances in Series and in Parallel\u003c\/p\u003e \u003cp\u003eFlow May Be Laminar or Turbulent\u003c\/p\u003e \u003cp\u003eShear Stress on the Vessel Wall\u003c\/p\u003e \u003cp\u003eRheologic Properties of Blood\u003c\/p\u003e \u003cp\u003eSummary\u003c\/p\u003e \u003cp\u003eCase 6-1\u003c\/p\u003e \u003cb\u003e \u003c\/b\u003e\u003cp\u003eChapter 7 \u003cb\u003eTHE ARTERIAL SYSTEM\u003c\/b\u003e\u003c\/p\u003e \u003cp\u003eThe Hydraulic Filter Converts Pulsatile Flow to Steady Flow\u003c\/p\u003e \u003cp\u003eArterial Elasticity Compensates for the Intermittent Flow Delivered by the Heart\u003c\/p\u003e \u003cp\u003eThe Arterial Blood Pressure Is Determined by Physical and Physiological Factors\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eMean Arterial Pressure\u003c\/p\u003e \u003cp\u003eCardiac Output\u003c\/p\u003e \u003cp\u003ePeripheral Resistance\u003c\/p\u003e \u003cp\u003ePulse Pressure\u003c\/p\u003e \u003cp\u003eStroke Volume\u003c\/p\u003e \u003cp\u003eArterial Compliance\u003c\/p\u003e \u003cp\u003eTotal Peripheral Resistance and Arterial Diastolic Pressure\u003c\/p\u003e \u003cp\u003eThe Pressure Curves Change in Arteries at Different Distances from the Heart\u003c\/p\u003e \u003cp\u003eBlood Pressure Is Measured by a Sphygmomanometer in Human Patients\u003c\/p\u003e \u003cp\u003eSummary\u003c\/p\u003e \u003cp\u003eCase 7-1\u003c\/p\u003e \u003cb\u003e \u003c\/b\u003e\u003cp\u003eChapter 8 \u003cb\u003eThe MICROCIRCULATION AND LYMPHATICS\u003c\/b\u003e\u003c\/p\u003e \u003cp\u003eFunctional Anatomy\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eArterioles Are the Stopcocks of the Circulation\u003c\/p\u003e \u003cp\u003eCapillaries Permit the Exchange of Water, Solutes, and Gases\u003c\/p\u003e \u003cp\u003eThe Law of Laplace Explains How Capillaries Can Withstand High Intravascular Pressures\u003c\/p\u003e \u003cp\u003eThe Endothelium Plays an Active Role in Regulating the Microcirculation\u003c\/p\u003e \u003cp\u003eThe Endothelium is at the Center of Flow-Initiated Mechanotransduction\u003c\/p\u003e \u003cp\u003eThe Endothelium Plays a Passive Role in Transcapillary Exchange\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eDiffusion Is the Most Important Means of Water and Solute Transfer Across the Endothelium\u003c\/p\u003e \u003cp\u003eDiffusion of Lipid-Insoluble Molecules Is Restricted to the Pores\u003c\/p\u003e \u003cp\u003eLipid-Soluble Molecules Pass Directly Through the Lipid Membranes of the Endothelium and the Pores\u003c\/p\u003e \u003cp\u003eCapillary Filtration Is Regulated by the Hydrostatic and Osmotic Forces Across the Endothelium\u003c\/p\u003e \u003cp\u003eBalance of Hydrostatic and Osmotic Forces\u003c\/p\u003e \u003cp\u003eThe Capillary Filtration Coefficient Provides a Method to Estimate the Rate of Fluid Movement Across the Endothelium\u003c\/p\u003e \u003cp\u003eHypoxia-inducible factor(s) and angiogenesis\u003c\/p\u003e \u003cp\u003ePinocytosis Enables Large Molecules to Cross the Endothelium\u003c\/p\u003e \u003cp\u003eThe Lymphatics Return the Fluid and Solutes That Escape Through the Endothelium to the Circulating Blood\u003c\/p\u003e \u003cp\u003eSummary\u003c\/p\u003e \u003cp\u003eCase 8-1\u003c\/p\u003e \u003cp\u003eCase 8-2\u003c\/p\u003e \u003cb\u003e \u003c\/b\u003e\u003cp\u003eChapter 9 \u003cb\u003eThe PERIPHERAL CIRCULATION AND ITS CONTROL\u003c\/b\u003e\u003c\/p\u003e \u003cp\u003eThe Functions of the Heart and Large Blood Vessels\u003c\/p\u003e \u003cp\u003eContraction and Relaxation of Arteriolar Vascular Smooth Muscle Regulate Peripheral Blood Flow\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eCytoplasmic Ca\u003csup\u003e++\u003c\/sup\u003e Is Regulated to Control Contraction, via MLCK\u003c\/p\u003e \u003cp\u003eContraction Is Controlled by Excitation-Contraction Coupling and\/or Pharmacomechanical Coupling\u003c\/p\u003e \u003cp\u003eControl of Vascular Tone by Catecholamines\u003c\/p\u003e \u003cp\u003eControl of Vascular Contraction by Other Hormones, Other Neurotransmitters, and Autocoids\u003c\/p\u003e \u003cp\u003eIntrinsic Control of Peripheral Blood Flow\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eAutoregulation and the Myogenic Mechanism Tend to Keep Blood Flow Constant\u003c\/p\u003e \u003cp\u003eThe Endothelium Actively Regulates Blood Flow\u003c\/p\u003e \u003cp\u003eTissue Metabolic Activity Is the Main Factor in the Local Regulation of Blood Flow\u003c\/p\u003e \u003cp\u003eExtrinsic Control of Peripheral Blood Flow Is Mediated Mainly by the Sympathetic Nervous System\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eImpulses That Arise in the Medulla Descend in the Sympathetic Nerves to Increase Vascular Resistance\u003c\/p\u003e \u003cp\u003eSympathetic Nerves Regulate the Contractile State of the Resistance and Capacitance Vessels\u003c\/p\u003e \u003cp\u003eThe Parasympathetic Nervous System Innervates Blood Vessels Only in the Cranial and Sacral Regions of the Body\u003c\/p\u003e \u003cp\u003eEpinephrine and Norepinephrine Are the Main Humoral Factors That Affect Vascular Resistance\u003c\/p\u003e \u003cp\u003eThe Vascular Reflexes Are Responsible for Rapid Adjustments of Blood Pressure\u003c\/p\u003e \u003cp\u003eThe Peripheral Chemoreceptors Are Stimulated by Decreases in Blood Oxygen Tension and pH and by Increases in Carbon Dioxide Tension\u003c\/p\u003e \u003cp\u003eThe Central Chemoreceptors Are Sensitive to Changes in Paco\u003csub\u003e2\u003c\/sub\u003e\u003c\/p\u003e \u003cp\u003eOther Vascular Reflexes\u003c\/p\u003e \u003cp\u003eBalance Between Extrinsic and Intrinsic Factors in Regulation of Peripheral Blood Flow\u003c\/p\u003e \u003cp\u003eSummary\u003c\/p\u003e \u003cp\u003eCase 9-1\u003c\/p\u003e \u003cb\u003e \u003c\/b\u003e\u003cp\u003eChapter 10 \u003cb\u003eCONTROL OF CARDIAC OUTPUT: COUPLING OF HEART AND BLOOD VESSELS\u003c\/b\u003e\u003c\/p\u003e \u003cp\u003eFactors Controlling Cardiac Output\u003c\/p\u003e \u003cp\u003eThe Cardiac Function Curve Relates Central Venous Pressure (Preload) to Cardiac Output\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003ePreload or Filling Pressure of the Heart\u003c\/p\u003e \u003cp\u003eCardiac Function Curve\u003c\/p\u003e \u003cp\u003eFactors That Change the Cardiac Function Curve\u003c\/p\u003e \u003cp\u003eThe Vascular Function Curve Relates Central Venous Pressure to Cardiac Output\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eMathematical Analysis of the Vascular Function Curve\u003c\/p\u003e \u003cp\u003eVenous Pressure Depends on Cardiac Output\u003c\/p\u003e \u003cp\u003eBlood Volume\u003c\/p\u003e \u003cp\u003eVenomotor Tone\u003c\/p\u003e \u003cp\u003eBlood Reservoirs\u003c\/p\u003e \u003cp\u003ePeripheral Resistance\u003c\/p\u003e \u003cp\u003eCardiac Output and Venous Return Are Closely Associated\u003c\/p\u003e \u003cp\u003eThe Heart and Vasculature Are Coupled Functionally\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eMyocardial Contractility\u003c\/p\u003e \u003cp\u003eBlood Volume\u003c\/p\u003e \u003cp\u003ePeripheral Resistance\u003c\/p\u003e \u003cp\u003eThe Right Ventricle Regulates Not Only Pulmonary Blood Flow but Also Central Venous Pressure\u003c\/p\u003e \u003cp\u003eHeart Rate Has Ambivalent Effects on Cardiac Output\u003c\/p\u003e \u003cp\u003eAncillary Factors Affect the Venous System and Cardiac Output\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eGravity\u003c\/p\u003e \u003cp\u003eMuscular Activity and Venous Valves\u003c\/p\u003e \u003cp\u003eRespiratory Activity\u003c\/p\u003e \u003cp\u003eArtificial Respiration\u003c\/p\u003e \u003cp\u003eSummary\u003c\/p\u003e \u003cp\u003eCase 10-1\u003c\/p\u003e \u003cp\u003eChapter 11 \u003cb\u003eCORONARY CIRCULATION\u003c\/b\u003e\u003c\/p\u003e \u003cp\u003eFunctional Anatomy of the Coronary Vessels\u003c\/p\u003e \u003cp\u003eCoronary Blood Flow Is Regulated by Physical, Neural, and Metabolic Factors\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003ePhysical Factors\u003c\/p\u003e \u003cp\u003eNeural and Neurohumoral Factors\u003c\/p\u003e \u003cp\u003eMetabolic Factors\u003c\/p\u003e \u003cp\u003eDiminished Coronary Blood Flow Impairs Cardiac Function\u003c\/p\u003e \u003cp\u003eEnergy Substrate Metabolism During Ischemia\u003c\/p\u003e \u003cp\u003eCoronary Collateral Vessels Develop in Response to Impairment of Coronary Blood Flow\u003c\/p\u003e \u003cp\u003eSummary\u003c\/p\u003e \u003cp\u003eCase 11-1\u003c\/p\u003e \u003cb\u003e \u003c\/b\u003e\u003cp\u003eChapter 12 \u003cb\u003eSPECIAL CIRCULATIONS\u003c\/b\u003e\u003c\/p\u003e \u003cp\u003eCutaneous Circulation\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eSkin Blood Flow Is Regulated Mainly by the Sympathetic Nervous System\u003c\/p\u003e \u003cp\u003eAmbient Temperature and Body Temperature Play Important Roles in the Regulation of Skin Blood Flow\u003c\/p\u003e \u003cp\u003eSkin Color Depends on the Volume and Flow of Blood in the Skin and on the Amount of O\u003csub\u003e2\u003c\/sub\u003e Bound to Hemoglobin\u003c\/p\u003e \u003cp\u003eSkeletal Muscle Circulation\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eRegulation of Skeletal Muscle Circulation\u003c\/p\u003e \u003cp\u003eCerebral Circulation\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eLocal Factors Predominate in the Regulation of Cerebral Blood Flow\u003c\/p\u003e \u003cp\u003eThe Pulmonary and Systemic Circulations Are in Series with Each Other\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eFunctional Anatomy\u003c\/p\u003e \u003cp\u003ePulmonary Hemodynamics\u003c\/p\u003e \u003cp\u003eRegulation of the Pulmonary Circulation\u003c\/p\u003e \u003cp\u003eThe Renal Circulation Affects the Cardiac Output\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eAnatomy\u003c\/p\u003e \u003cp\u003eRenal Hemodynamics\u003c\/p\u003e \u003cp\u003eThe Renal Circulation Is Regulated by Intrinsic Mechanisms\u003c\/p\u003e \u003cp\u003eThe Splanchnic Circulation Provides Blood Flow to the Gastrointestinal Tract, Liver, Spleen, and Pancreas\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eIntestinal Circulation\u003c\/p\u003e \u003cp\u003eHepatic Circulation\u003c\/p\u003e \u003cp\u003eFetal Circulation\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eChanges in the Circulatory System at Birth\u003c\/p\u003e \u003cp\u003eSummary\u003c\/p\u003e \u003cp\u003eCase 12-1\u003c\/p\u003e \u003cp\u003eCase 12-2\u003c\/p\u003e \u003cp\u003eCase 12-3\u003c\/p\u003e \u003cb\u003e \u003c\/b\u003e\u003cp\u003eChapter 13 \u003cb\u003eINTERPLAY OF CENTRAL AND PERIPHERAL FACTORS THAT CONTROL THE CIRCULATION\u003c\/b\u003e\u003c\/p\u003e \u003cp\u003eExercise\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eMild to Moderate Exercise\u003c\/p\u003e \u003cp\u003eSevere Exercise\u003c\/p\u003e \u003cp\u003ePostexercise Recovery\u003c\/p\u003e \u003cp\u003eLimits of Exercise Performance\u003c\/p\u003e \u003cp\u003ePhysical Training and Conditioning\u003c\/p\u003e \u003cp\u003eHemorrhage\u003c\/p\u003e\n\u003ci\u003e \u003c\/i\u003e\u003cp\u003eHemorrhage Evokes Compensatory and Decompensatory Effects on the Arterial Blood Pressure\u003c\/p\u003e \u003cp\u003eThe Compensatory Mechanisms Are Neural and Humoral\u003c\/p\u003e \u003cp\u003eThe Decompensatory Mechanisms Are Mainly Humoral, Cardiac, and Hematologic\u003c\/p\u003e \u003cp\u003eThe Positive and Negative Feedback Mechanisms Interact\u003c\/p\u003e \u003cp\u003eSummary\u003c\/p\u003e \u003cp\u003eCase 13-1\u003c\/p\u003e \u003cp\u003eCase 13-2\u003c\/p\u003e \u003cb\u003e \u003c\/b\u003e\u003cp\u003eAppendix A: End-of-Chapter CASE STUDY ANSWERS\u003c\/p\u003e \u003cp\u003eAppendix B: Comprehensive Examination\u003c\/p\u003e \u003c\/font\u003e\u003c\/p\u003e\r\n\r\n\u003cp\u003e\u003cfont size=\"3\"\u003eSubject Areas: Physiology [\u003ca title=\"See our other books on Physiology\" href=\"https:\/\/freshlyprintedbooks.co.uk\/search?q=%22Physiology%20%5BMFG%5D%22\"\u003eMFG\u003c\/a\u003e]\u003c\/font\u003e\u003c\/p\u003e\r\n\r\n\r\n\u003c\/font\u003e","brand":"Elsevier","offers":[{"title":"Default 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